Quantum radiation pressure on a moving mirror at finite temperature

We compute the radiation pressure force on a moving mirror, in the nonrelativistic approximation, assuming the field to be at temperature $T.$ At high temperature, the force has a dissipative component proportional to the mirror velocity, which results from Doppler shift of the reflected thermal photons. In the case of a scalar field, the force has also a dispersive component associated to a mass correction. In the electromagnetic case, the separate contributions to the mass correction from the two polarizations cancel. We also derive explicit results in the low temperature regime, and present numerical results for the general case. As an application, we compute the dissipation and decoherence rates for a mirror in a harmonic potential well.Comment: Figure 3 replaced, changes mainly in Sections IV and V, new appendix introduced. To appear in Physical Review

Effects of cholinergic stimulation with pyridostigmine bromide on chronic chagasic cardiomyopathic mice

The aim of the present study was to assess the effects of an anticholinesterase agent, pyridostigmine bromide (Pyrido), on experimental chronic Chagas heart disease in mice. To this end, male C57BL/6J mice noninfected (control:Con) or chronically infected (5 months) with Trypanosoma cruzi (chagasic:Chg) were treated or not (NT) with Pyrido for one month. At the end of this period, electrocardiogram (ECG); cardiac autonomic function; heart histopathology; serum cytokines; and the presence of blood and tissue parasites by means of immunohistochemistry and PCR were assessed. In NT-Chg mice, significant changes in the electrocardiographic, autonomic, and cardiac histopathological profiles were observed confirming a chronic inflammatory response. Treatment with Pyrido in Chagasic mice caused a significant reduction of myocardial inflammatory infiltration, fibrosis, and hypertrophy, which was accompanied by a decrease in serum levels of IFN\u3b3 with no change in IL-10 levels, suggesting a shift of immune response toward an anti-inflammatory profile. Lower nondifferent numbers of parasite DNA copies were observed in both treated and nontreated chagasic mice. In conclusion, our findings confirm the marked neuroimmunomodulatory role played by the parasympathetic autonomic nervous system in the evolution of the inflammatory-immune response to T. cruzi during experimental chronic Chagas heart disease in mice